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The term “peptic ulcer” refers to ulcers that occur in the stomach (gastric ulcer) or the first part of the small intestine (duodenal ulcer).

Duodenal ulcers are more common, and the frequency of their occurrence in the adult population of the United States is 6-12%. In other words, there is clinical evidence of duodenal ulcer in approximately 10% of the US population at some point in their lives. Duodenal ulcer occur 4 times more often in men than in women, and, in General, is 4-5 times more common than stomach ulcers. Although the symptoms of peptic ulcer may be absent or quite vague, most peptic ulcers are associated with abdominal discomfort, celebrate 45-60 minutes after a meal or at night. In the typical case, pain is described as aching, burning, cramping, severe pain or “heartburn”. Eating or using antacids usually results in substantial relief of symptoms.

 

What causes ulcers?

 

Although duodenal ulcer and gastric occur in different places in the body, they have, apparently, similar mechanisms.

 

In particular, the development of duodenal ulcers or gastric ulcer is a result of the impact of some factors that Deplete the protective factors of the membranes of the stomach and duodenum.

 

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In the past the main attention was paid mainly to acid secretions of the stomach, which were considered as the main cause of ulcers of the stomach and duodenum.

 

However, recently the focus has shifted to the bacteria Helicobacter pylori (Helicobacter pylori) and nonsteroidal anti-inflammatory drugs (NSAIDs) such as aspirin and ibuprofen.

 

Stomach acid is extremely corrosive. Due to the high acidity (pH 1 to 3) stomach acid would immediately corrode the skin and create ulcers.

 

To protect against ulcers in the mucosa of the stomach and small intestine have a layer of mucin.

 

In addition, the constant renewal of intestinal cells and the secretion of substances that neutralize acid upon contact with the stomach and membranes of the intestines, also protect from the formation of ulcers.

 

Acid is designed to digest food that we eat, not the stomach or small intestine.

 

Contrary to popular belief, excessive secretion in the development of gastric acid is rarely a factor in causing stomach ulcers.

 

In fact, patients with stomach ulcers, as a rule, stand normal or even reduced levels of stomach acid.

 

In turn, almost half of the patients with duodenal ulcer observed increased production of stomach acid.

 

This increase may be due to the increase in the number of cells that produce acid are called parietal cells.

 

In the study of patients with duodenal ulcers in the group noticed that they have twice the parietal cells in the stomach compared to people without ulcers.

 

Even with the increase the production of stomach acid under normal conditions, the protective shell would prevent the formation of ulcers of the stomach or duodenum. However, in violation of the integrity of these protective sheaths can be formed ulcer.

 

Loss of integrity may be the effect of Helicobacter pylori (H. pylori), aspirin and other nonsteroidal anti-inflammatory drugs (NSAIDs), Smoking, alcohol, nutrient deficiency, stress and many other factors.

 

 

 

What is the best natural cure for ulcers?

 

This is a special licorice extract known as DGL.

 

Licorice has long been considered a great remedy for peptic ulcer.

 

However, to avoid side effects of any component of licorice, as glitzirrizinova acid (in some cases it causes a rise in blood pressure), developed a procedure of removing this compound from licorice and create deglycyrrhizinated licorice (DGL). The result is a very successful anti-ulcer agent without any known side effects.

 

How does DGL?

 

The nature of the mechanism of action of DGL is as follows: it stimulates and/or accelerates the action of protective factors that counteract the formation of ulcers.

 

This mechanism is very different from the effects of antacids and drugs such as Tagamet, Zantac, Pepcid, Prevacid and Prilosec, which act by neutralizing or suppressing gastric acid.

 

Raises an obvious question related to DGL: “does DGL have any effect on Helicobacter pylori?”

 

It seems that this question needs to be answered in the affirmative because DGL includes several flavonoids that have been shown to inhibit Helicobacter pylori.12.

 

What DGL is different from antacids and drugs such Tagamet and Zantac?

 

Numerous studies over the years have shown that DGL is an effective anti-ulcer compound.

 

In several comparative studies in which drugs were compared with DGL in pairs, it was found that DGL is more effective of Tagamet, Santaka and antacids for short-term and maintenance therapy of peptic ulcers.

 

However, while these drugs can cause significant side effects, DGL is extremely safe and is many times cheaper.

 

What studies have shown effects of DGL for gastric ulcer?

 

Were obtained very good results. For example, in the study of the use of DGL in treating gastric ulcers 33 patients with gastric ulcer received either DGL (760 mg three times daily) or placebo for one month.

 

The study noted a more significant decrease in the size of ulcers in the DGL group (78%) than in the placebo group (34%). Full recovery occurred in 44% of patients receiving DGL, and only 6% patients of the placebo group.

 

Subsequent studies have shown that DGL is as effective as Tagamet and Zantac under short-term and maintenance therapy of gastric ulcers.

 

For example, when compared with Tagametum 100 patients received either DGL (760 mg, 3 times per day between meals), or Tagamet (200 mg, 3 times daily and 400 mg at bedtime).

 

The percentage of ulcers healed after 6 and 12 weeks was similar in both groups. However, Tagamet is somewhat toxic, but DGL is a completely safe to use.

 

The occurrence of gastric ulcers is often the result of alcohol consumption, aspirin or other nonsteroidal anti-inflammatory drugs, caffeine, as well as the impact of other factors that violate the integrity of the gastric mucosa.

 

As DGL has been shown to reduce gastric bleeding caused by aspirin, it is strongly recommended to take for prevention of stomach ulcers in patients who need long-term treatment with drugs that cause ulcer, such as aspirin, other NSAIDs and corticosteroids.

 

What is the effect of DGL for ulcers of the duodenum?

 

DGL is also effective in duodenal ulcers. This is perhaps best illustrated by one study of patients with severe duodenal ulcers.

 

In the study, forty patients with chronic duodenal ulcers with disease duration from 4 to 12 years and more than 6 relapses during the previous year received DGL.

 

All patients were sent for surgery because of relentless pain, sometimes with frequent vomiting, despite treatment with bed rest, antacids, and powerful drugs.

 

Half of the patients received 3 grams of DGL daily for 8 weeks; the other half received 4.5 grams per day for 16 weeks.

 

All 40 patients experienced significant improvement, usually within 5-7 days, and none of them needed surgical intervention during follow-up for 1 year.

 

Although both doses were effective, the higher dose was significantly more effective than low dose.

 

In another more recent study, the therapeutic effect of DGL was compared with the therapeutic effect of antacids or cimetidine from 874 patients with confirmed chronic duodenal ulcers.

 

89 91% of all ulcers healed within 12 weeks was not observed any significant differences in speed of healing in different groups.

 

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However, subjects in the DGL group had fewer relapses (8.2 per cent) than in those receiving cimetidine (12.9 per cent) or antacids (16.4 per cent).

 

These results in combination with DGL protective effects, suggest that DGL is the best treatment of duodenal ulcers.

 

How do I take DGL?

 

Standard dosage DGL in acute cases is two to four chewable tablets of 400 mg between meals or 20 minutes before a meal.

 

Dosage in less severe chronic cases, and the supportive dosage is one to two tablets 20 minutes before meals.

 

Welcome DGL after meals leads to bad results.

 

Treatment with DGL should be continued for 8-16 weeks after a complete therapeutic response.

 

Apparently, to ensure the effectiveness of DGL in the healing of peptic ulcers, it needs to mix with saliva.

 

DGL may promote the release of salivary compounds that stimulate growth and cell regeneration of the stomach and intestines.

 

It should also be noted that the DGL in capsule form is ineffective.

 

Antacids seem to help alleviate my symptoms. Do I need to continue to use them or they will reduce the effectiveness of DGL?

 

Antacids can be used as part of the initial treatment to alleviate symptoms.

 

All antacids are relatively safe for occasional use, but I strongly recommend avoiding antacids with aluminum.

 

I suggest to follow the instructions on the labels and avoid regular or excessive use of antacids.

 

Regular intake of antacids can lead to malabsorption of nutrients, disorders of the bowel, kidney stones and other side effects.published econet.ru.

©Dr. Michael Murray

 

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